Like Mother, Like Daughter! Kate Hudson’s Daughter ‘Wears So Many Hats'

Kate Hudson loves her little ones! The actress became a mom in 2004 and has been gushing about her family ever since.

The Almost Famous star most recently welcomed her daughter, Rani, in October 2018 with her boyfriend, Danny Fujikawa. “She’s here,” the Fabletics creator wrote on Instagram at the time. “We have decided to name our daughter Rani (pronounced Ronnie) after her grandfather, Ron Fujikawa. Ron was the most special man who we all miss dearly. To name her after him is an honor. Everyone is doing well and happy as can be. Our family thanks you for all the love and blessings that have been sent our way and we send ours right back.”

This came six months after Hudson announced that she and Fujikawa were expecting their first child together. “We have been trying to keep this pregnancy under the radar for as long as possible but I’m a poppin now!” the Los Angeles native wrote on Instagram in April 2018. “And it’s too darn challenging to hide, and frankly hiding is more exhausting then just coming out with it! My kids, Danny, myself and the entire family are crazy excited! A little girl on the way.”

The Golden Globe winner welcomed Ryder in 2004 with her ex-husband, Chris Robinson, followed by Bingham seven years later with her ex-fiancé, Matt Bellamy.

In April 2019, a source told Us Weekly about her coparenting relationships with the Black Crowes singer and the Muse frontman. “[She] sees Matt a lot with Bing, who also has separate time with his dad,” the insider explained to Us at the time. “Chris lives in northern California but comes down to visit because Kate supports Ryder having a positive relationship with his dad.”

Keep scrolling for a look at the Pretty Fun author’s best quotes about her family, from her parenting style to her post-baby body journey.

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What will Australia’s COVID vaccination program look like? 4 key questions answered

The Pfizer/BioNTech, Moderna and AstraZeneca/University of Oxford groups have all recently announced their COVID vaccine candidates have demonstrated high levels of efficacy in phase 3 trials. These developments have focused attention on how a COVID vaccine might be rolled out in Australia.

It’s important to emphasize these trials have not yet been completed, and we only have a few headline results. But the information we do have is promising, and pending scrutiny from the Therapeutic Goods Administration (TGA), it’s looking increasingly hopeful we’ll have several COVID vaccines available in Australia during 2021.

To make this happen, a lot goes on behind the scenes. Australia’s national strategy for the delivery of a COVID vaccine encompasses the whole process: from research and development, to purchase and manufacturing, to international partnerships, to regulation and safety, to administration and monitoring.

Here’s a summary of some of the things you might be wondering about how this all works.

1. Which vaccines will we get?

Currently, there are more than 200 vaccine candidates around the world, 48 of which are in clinical trials.

To ensure Australians will have access to COVID-19 vaccines, the federal government has established agreements with suppliers of four of the most promising vaccine candidates. The vaccines have been carefully assessed on advice from a Science and Industry Technical Advisory Group.

So far, the vaccines we’ve signed up to include the AstraZeneca/University of Oxford vaccine, the CSL/University of Queensland vaccine, the Novavax vaccine, and the Pfizer/BioNTech vaccine.

These agreements will only progress should the vaccines prove safe and effective, as assessed by the TGA, which will look at the quality of the vaccine, the degree of protection it offers, and its safety.

In addition to the four pre-purchase agreements, the federal government has signed up to the global vaccine initiative COVAX, which supports vaccines for all participating countries and grants us access to a range of additional leading candidates.

2. Once we get a vaccine, who will receive it first?

The intent is that a vaccine will eventually be available for anyone who wants to be vaccinated. But it’s likely the initial supply will be limited, so we’ll need to make decisions around which groups will receive the vaccine first.

This will depend on the characteristics of the available vaccines as well as principles we use to define priority populations. These include using vaccines in those who will benefit the most, ensuring equitable access, and reciprocity (the obligation to those who bear additional risks as part of the COVID-19 response).

Australia’s COVID-19 vaccination policy sets out target groups including people who are at higher risk of severe disease and death from COVID-19 (especially older people), those at greater risk of exposure and transmission (health-care and aged-care workers) and other essential workers required to maintain the functioning of society (such as police).

The exact priority order may depend on whether the vaccine works as well in older people, whether it protects against infection (and therefore transmission) or only severe disease, and where infections are occurring when vaccines become available.

3. How will people get it?

We’re likely to need a range of vaccination providers and sites to ensure timely access for the population.

Distribution may be complicated by different storage requirements—for example, the Pfizer vaccine needs to be stored at -70℃. While this might sound like a major hurdle, the Ebola vaccine required similar storage conditions and was successfully delivered in West Africa. But this is something we need to take into account.

People involved in vaccine distribution and administration will need additional training in the specifics of these new vaccines. This workforce will be crucial to a successful COVID-19 vaccine program, particularly if we need to set up additional clinics.

As it’s likely we’ll need two vaccine doses, keeping records on who has received a vaccine so reminders can be sent for the second dose will be important.

4. How will we know if it’s safe?

Current clinical trials are including up to 30,000-40,000 participants, of which roughly half receive the vaccine.

Studies of this size are sufficient to identify common adverse events (like a sore arm or fever, which we’re seeing reported in some patients). But to pick up serious but very rare side effects, ongoing monitoring of vaccine safety will be important.

Existing national and state-based surveillance programs will monitor the safety of COVID-19 vaccines. For example, the AusVaxSafety system sends text messages to those who have received vaccines to check on side effects, and SAFEVAC is a network of experts who assess reports of adverse events and can provide clinical advice.

Using the Australian Immunization Register to record COVID vaccinations will also be crucial to monitoring safety and effectiveness.

Finally, we’ll need to communicate with the public about what we know, as well as the uncertainties, as a vaccine is rolled out. This involves identifying which groups need information, developing and disseminating evidence-based resources, and supporting health-care providers to facilitate discussions with patients. We’ll also need strategies to manage negative messaging and misinformation.

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What It Feels Like… to have incurable cancer

Most people born in the UK today can expect to live until they’re about 80.

Having been diagnosed with incurable breast cancer some 18 months ago, aged just 55, I’ll be lucky to make it to my 60s.

How do I deal with that knowledge? I find ways, otherwise I would go mad worrying about it all the time. 

I was first diagnosed with primary breast cancer in the summer of 2015 after I noticed a lump and other changes in my right breast. Over the following seven months, I had chemotherapy, followed by a mastectomy and the removal of the lymph nodes in my right armpit, and radiotherapy.

I finished active treatment in February 2016. At that point, you’re deemed to be ‘cancer-free’, but lots of people, including me, don’t like and don’t use that phrase.

We prefer the term ‘no evidence of disease’. Although treatment can get rid of all the cancer that can be found, there’s always a chance that undetected cells have or will spread to other areas of the body.

The cells lie dormant and, in some cases, turn up as active cancer later. This happened to me just over three years after my initial treatment finished.

It is known as secondary, metastatic, Stage IV, advanced or late-stage breast cancer. It can’t be cured.

For me, the initial breast cancer spread to my bones and bone marrow. I received my secondary cancer diagnosis after I followed up on some back pain I’d been having.

The later the stage of your initial diagnosis (basically how big the tumour is and whether it’s spread to the lymph nodes), the more likely it is that you’ll develop ‘secondaries’ down the line. I was Stage III when I was first diagnosed, so I knew I was high risk.

No-one recovers from secondary breast cancer. It can be treated but it will kill you eventually – unless you die of something else in the meantime.

Some women live for many years with the disease but data indicates that 50% die within three years of being diagnosed.

You have no way of knowing which camp you’ll fall into.

My oncologist told me a year ago that she’d be disappointed if I didn’t have at least another five years left. Initially, it was all I could think about.

I have a husband and two young-adult sons, and I was engulfed by a wave of sadness every time I heard one of my boys call me ‘mum’. I’d wonder how much longer I’d be around for them to be able to say that. It was the same with my husband calling my name. 

Now, rather than letting the sound of the words pain me, I let them sweep over me like a comforting blanket.

I try to focus on the present instead of on the future. ‘Never borrow tomorrow’s sorrow’ – this was the advice from a member of an invaluable online support group I’ve joined. It’s a good way of looking at it. 

Lots of people experience periods of intense sadness and are still happy in the long run. There are no guarantees in life for any of us, other than that we will all die at some point. That fact helps keep me grounded.

Sometimes if I’m out and about and I start thinking dark thoughts, I look at the people around me and think that any one of them could be dead tomorrow. That may sound odd, but I find it helpful. They’re not all walking about worrying about when and how death will come to them – so I don’t have to either .

I have to work hard at building up my resilience. That doesn’t mean you simply pretend everything’s fine when it’s clearly not – it isn’t just a case of having a positive attitude.

For me, resilience means accepting and acknowledging my situation, allowing myself to be angry and sad and scared, while at the same time knowing that those feelings needn’t take over and override every other emotion.

The facts are there, as a backdrop to my life. I can’t escape them, but I do try to ‘live alongside my cancer’, as one of my doctors puts it. That’s my aim, for me and my family. 

Both my husband and I have been open about the situation with our sons, as we don’t want them to worry that we’re hiding things from them. We keep them up to date with how my treatment is going, but I really hope that for most of the time, at least, my situation takes a back seat in their young lives. 

I also hope they feel they can talk about it – if not to us, then to whoever they think will be able to listen well and provide comfort.

At the moment I am in no pain from the disease. My hair gets thinner by the day, my nails are a mess and the occasional pain I get in my feet can stop me from sleeping. Other than that, I feel incredibly healthy. 

Coronavirus restrictions permitting, I swim, walk, cycle and play tennis. My immune system is weak due to the cancer itself and the treatment I’m on, so I do have to be careful. 

With regard to the pandemic, I’m far from reckless but I am out there, trying to make the most of things. Recently, I walked much of the Hadrian’s Wall Way, covering 68 of the 84 miles in six days, and my husband and I have just spent 10 fabulous days travelling around northern Greece.

I currently take oral chemotherapy tablets in the morning and evening, one week on, one week off. I have a monthly injection of a bone-strengthening drug as well as regular scans. I have monthly blood tests, then I see my consultant to discuss the results and how I’m managing any treatment side effects.

At the moment, the cancer is restricted to my bones and bone marrow. At some point, it is likely to spread elsewhere. Things can change from one month to the next.

There’s no right or wrong approach to dealing with a diagnosis such as mine. At the moment, acceptance and pragmatism work for me. It’s not easy but I carry on, taking each day as it comes and dealing with the good and the bad.

I seize the moment when I can, give thanks and relish the life I have – as well as the many, many wonderful people I have in it.

You can find Maureen’s blog here. The lead photo was taken by Mallorca Cycling Photos.

The Institute of Cancer Research, London, is currently fundraising to complete its new Centre for Cancer Drug Discovery, which will house the world’s first drug discovery programme focusing on tackling cancer’s ability to evolve and develop resistance to drugs. You can support its appeal here.

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In this exciting new series from, What It Feels Like… not only shares one person’s moving story, but also the details and emotions entwined within it, to allow readers a true insight into their life changing experience.

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Just Like Dad! Tiger Woods' Son Charlie, 11, Wins Junior Golf Tournament

Like father, like son! Tiger Woods’ 11-year-old son, Charlie, is following in his father’s footsteps.

Tiger Woods and Ex-Wife Elin Nordegren’s Coparenting Quotes

The preteen competed at Hammock Creek Golf Club in Palm City, Florida, earlier this month. Charlie played in a U.S. Kids Golf-sanctioned event and rose to the top of the leaderboard in the boys’ 11-year-old division.

The little one shot three-under 33 across nine holes, finishing with three birdies and zero bogeys, according to U.S. Kids Golf’s stats.

Celebrities and Their Look-Alike Kids

When Charlie competed at a junior golf event in South Florida in January, Woods, 44, caddied for his son. “Tiger Woods’ son at age 10 hits it better than I have my entire 27 years of existence,” one Twitter user captioned a video of Charlie’s golfing skills.

The professional golfer shares his son with ex-wife Elin Nordegren, as well as their 13-year-old daughter, Sam. Following the How I Play Golf author’s 2009 cheating scandal, the former couple have worked hard to amicably coparent their kids.

“Having the relationship that I have now with her is fantastic,” the athlete told Time in December 2015. “She’s one of my best friends. We’re able to pick up the phone, and we talk to each other all the time. We both know that the most important things in our lives are our kids. I wish I would have known that back then.”

The California native added at the time: “I’ve taken the initiative with the kids, and told them upfront, ‘Guys, the reason why we’re not in the same house, why we don’t live under the same roof, Mommy and Daddy, is because Daddy made some mistakes.’”

Putting the Kids 1st! These Former Celeb Couples Are Crushing Coparenting

Nordegren, 40, became a mom for the third time in October 2019 when she welcomed her and boyfriend Jordan Cameron’s first child together.

Us Weekly confirmed the Swedish star’s pregnancy in June 2019. Nordegren was “very happy” about the news, a source said at the time, adding, “Elin maintains a low profile and has a normal, boring life. Florida allows her to live that quiet life. … She’s surprised that people care that she’s pregnant.”

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Sperm don’t swim anything like we thought they did, new study finds

Under a microscope, human sperm seem to swim like wiggling eels, tails gyrating to and fro as they seek an egg to fertilize. 

But now, new 3D microscopy and high-speed video reveal that sperm don’t swim in this simple, symmetrical motion at all. Instead, they move with a rollicking spin that compensates for the fact that their tails actually beat only to one side. 

“It’s almost like if you’re a swimmer, but you could only wiggle your leg to one side,” said study author Hermes Gadêlha, a mathematician at the University of Bristol in the U.K. “If you did this in a swimming pool and you only did this to one side, you would always swim in circles. … Nature in its wisdom came [up] with a very complex, ingenious way to go forward.” 

Strange swimmers

The first person to observe human sperm close up was Antonie van Leeuwenhoek, a Dutch scientist known as the father of microbiology. In 1677, van Leeuwenhoek turned his newly developed microscope toward his own semen, seeing for the first time that the fluid was filled with tiny, wiggling cells. 

Under a 2D microscope, it was clear that the sperm were propelled by tails, which seemed to wiggle side-to-side as the sperm head rotated. For the next 343 years, this was the understanding of how human sperm moved. 

“[M]any scientists have postulated that there is likely to be a very important 3D element to how the sperm tail moves, but to date we have not had the technology to reliably make such measurements,” said Allan Pacey, a professor of andrology at the University of Sheffield in England, who was not involved in the research. 

The new research is thus a “significant step forward,” Pacey wrote in an email to Live Science. 

Gadêlha and his colleagues at the Universidad Nacional Autónoma de México started the research out of “blue-sky exploration,” Gadêlha said. Using microscopy techniques that allow for imaging in three dimensions and a high-speed camera that can capture 55,000 frames per second, they recorded human sperm swimming on a microscope slide. 

“What we found was something utterly surprising, because it completely broke with our belief system,” Gadêlha told Live Science. 

The sperm tails weren’t wiggling, whip-like, side-to-side. Instead, they could only beat in one direction. In order to wring forward motion out of this asymmetrical tail movement, the sperm head rotated with a jittery motion at the same time that the tail rotated.The head rotation and the tail are actually two separate movements controlled by two different cellular mechanisms, Gadêlha said. But when they combine, the result is something like a spinning otter or a rotating drill bit. Over the course of a 360-degree rotation, the one-side tail movement evens out, adding up to forward propulsion.

“The sperm is not even swimming, the sperm is drilling into the fluid,” Gadêlha said. 

The researchers published their findings today (July 31) in the journal Science Advances.

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Asymmetry and fertility

In technical terms, how the sperm moves is called precession, meaning it rotates around an axis, but that axis of rotation is changing. The planets do this in their rotational journeys around the sun, but a more familiar example might be a spinning top, which wobbles and dances about the floor as it rotates on its tip. 

“It’s important to note that on their journey to the egg that sperm will swim through a much more complex environment than the drop of fluid in which they were observed for this study,” Pacey said. “In the woman’s body, they will have to swim in narrow channels of very sticky fluid in the cervix, walls of undulating cells in the fallopian tubes, as well have to cope with muscular contractions and fluid being pushed along (by the wafting tops of cells called cilia) in the opposite direction to where they want to go. However, if they are indeed able to drill their way forward, I can now see in much better clarity how sperm might cope with this assault course in order to reach the egg and be able to get inside it,” Pacey said

Sperm motility, or ability to move, is one of the key metrics fertility doctors look at when assessing male fertility, Gadêlha said. The rolling of the sperm’s head isn’t currently considered in any of these metrics, but it’s possible that further study could reveal certain defects that disrupt this rotation, and thus stymy the sperm’s movement. 

Fertility clinics use 2D microscopy, and more work is needed to find out if 3D microscopy could benefit their analysis, Pacey said. 

“Certainly, any 3D approach would have to be quick, cheap and automated to have any clinical value,” he said. “But regardless of this, this paper is certainly a step in the right direction.”

Originally published in Live Science.

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